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Recently two tests were performed using very potent Anadenanthera colubrina seeds. In both tests 400 mg of seeds were used. This was two seeds in both test which were selected from many to come to this weight within +-5 mg.
No special treatment was performed on the seeds in the first test. The seeds were consumed orally after de-hulling them. In both cases 1100 mg of ginger was taken to try to stop the nausea. In the second test the seeds were ground with 1200 mg of calcium carbonate and a little water was then added, maybe 50 ml? It was then mixed for 8 hours overnight. After that, it was consumed as is.
So basically the only difference between test 1 and test 2 was that in test 2 the seeds were mixed with 3 times their weight in calcium carbonate and water for 8 hours.
In both cases AFOAF says the seeds were active in him at the dose used.
The effects between test 1 and test 2 were NOT THE SAME. They were very distinctly different. Both caused nausea despite taking the ginger. In test 2, AFOAF unfortunately vomited out some of the seed material, so he didn't get a complete dose.
Here's a rundown of the differences.
Test 1: visuals were less defined, more like outlines, sharp though. The tingling was sharp, edgy, like that of freebase bufotenine taken orally. There was a sour taste in the mouth. AFOAF felt a little sick, not just nausea. All of the effects were pretty much identical to taking 100 mg of freebase bufotenine orally.
Test 2: the visual effects were more like DMT, definitely more defined, smooth, soft. The tingling sensations were more like that experienced from elemicin, very pleasurable, smooth. There was no sick feeling other than the occasional nausea. The effects were very different from taking freebase bufotenine orally, but roughly equipotent to taking 100 mg of freebase bufotenine orally.
Why these seeds are more potent then bufotenine is, I'm not sure. Perhaps MAOIs are present in the seeds. The seeds at most contain 12% bufotenine. At 400 mg orally that's at most 48 mg bufotenine.
So what is going on here? Why are the effects of test 2 unlike freebase bufotenine and more like DMT? What accounts for this? The calcium carbonate is not just freebasing the bufotenine. I know. AFOAF has used freebase bufotenine orally many times. Something is changing. The bufotenine is altered by this somehow.